Hormonal Carcinogenesis V
Information gathered from cell-free systems, cell cultures, animal models, and human studies, together will (1) provide important insights to our understanding of hormonal cancer causation, development, and prevention; (2) be the primary objective of these Symposia.
Corporate Author: | |
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Other Authors: | , , , , |
Language: | English |
Published: |
New York, NY :
Springer New York : Imprint: Springer,
2008.
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Edition: | 1st ed. 2008. |
Series: | Advances in Experimental Medicine and Biology,
617 |
Subjects: | |
Online Access: | https://doi.org/10.1007/978-0-387-69080-3 |
Table of Contents:
- Symposium Address
- State of the Art Speakers
- Celluar Origins of Endocrine-related Cancers
- Mitotic Kinases, Centrosome Amplification, and Genomic Instability
- New Developments in Steroid Receptor Interactions
- Risk Assessment and Relevant Early Biomarkers
- Novel Strategies for Preventing and Treatment of Endocrine-related Cancers
- Hormone Dependency Versus Hormone Independency
- Emerging Fields: Hormones and Colorectal and Lung Cancers
- Communications: Session I
- Communications: Session I
- Communications: Session I
- Communications: Session I
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
- Communications: Session II: Cell/Molecular Biology and Hormone Metabolism.