Hormonal Carcinogenesis V

Information gathered from cell-free systems, cell cultures, animal models, and human studies, together will (1) provide important insights to our understanding of hormonal cancer causation, development, and prevention; (2) be the primary objective of these Symposia.

Corporate Author: SpringerLink (Online service)
Other Authors: Li, Jonathan J. (Editor, http://id.loc.gov/vocabulary/relators/edt), Li, Sara A. (Editor, http://id.loc.gov/vocabulary/relators/edt), Mohla, Suresh. (Editor, http://id.loc.gov/vocabulary/relators/edt), Rochefort, Henri. (Editor, http://id.loc.gov/vocabulary/relators/edt), Maudelonde, Thierry. (Editor, http://id.loc.gov/vocabulary/relators/edt)
Language:English
Published: New York, NY : Springer New York : Imprint: Springer, 2008.
Edition:1st ed. 2008.
Series:Advances in Experimental Medicine and Biology, 617
Subjects:
Online Access:https://doi.org/10.1007/978-0-387-69080-3
Table of Contents:
  • Symposium Address
  • State of the Art Speakers
  • Celluar Origins of Endocrine-related Cancers
  • Mitotic Kinases, Centrosome Amplification, and Genomic Instability
  • New Developments in Steroid Receptor Interactions
  • Risk Assessment and Relevant Early Biomarkers
  • Novel Strategies for Preventing and Treatment of Endocrine-related Cancers
  • Hormone Dependency Versus Hormone Independency
  • Emerging Fields: Hormones and Colorectal and Lung Cancers
  • Communications: Session I
  • Communications: Session I
  • Communications: Session I
  • Communications: Session I
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism
  • Communications: Session II: Cell/Molecular Biology and Hormone Metabolism.